There is strong evidence that excess abdominal fat, and particularly visceral fat, might impair immune system function. Inflammation and alterations in immune cell activity are only two parts of the intricate web that connects abdominal fat to the immune system.
First, inflammation causes the release of inflammatory chemicals, such as cytokines and adipokines, by metabolically active visceral fat.
The Role of Adipokines in Immune Function: Lipitin and Adiponectin: Two adipokines that are secreted by fat cells have the potential to impact how the immune system works.
Problems with immune cells, including macrophages and inflammatory adipose tissue, can develop when visceral fat attracts these cells.
Insulin Resistance: Its Effects on Immune Cells: Insulin resistance, which is commonly linked to obesity, can compromise the immune system's ability to fight off infections.
Deterioration of Immune Surveillance: T-Cell Dysfunction: Inflammation caused by obesity can cause T cells, an essential part of the immune system, to malfunction.
Obesity and extra abdominal fat may be linked to alterations in the gut microbiota, which can lead to compromised gut health and microbial imbalance.
Impact on immunological Function: Inflammation and immunological dysfunction may be exacerbated by gut health problems, which in turn affect the immune system.
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