It's correct. Belly fat, especially visceral fat around internal organs, is closely connected to insulin resistance and type 2 diabetes. This connection works:
Visceral fat releases free fatty acids into the circulation during metabolism. High quantities of free fatty acids can cause insulin resistance.
Adipokines from visceral fat cause inflammation. Chronic inflammation is thought to cause insulin resistance.
Hormonal imbalances: Excess abdominal fat can increase cortisol production. These hormonal changes can cause insulin resistance.
Fat cells release insulin-sensitizing and anti-inflammatory adiponectin. Adiponectin levels drop in those with significant abdominal fat, contributing to insulin resistance.
Non-alcoholic fatty liver disease, which causes fat buildup in organs other than fat tissue, increases insulin resistance and type 2 diabetes risk.
Insulin resistance develops when cells become less receptive to insulin, which regulates blood sugar. The pancreas generates more insulin to compensate for the decreased efficacy. Over time, pancreatic stress can cause type 2 diabetes, which raises blood sugar.
Lifestyle adjustments like diet, exercise, and weight management can reduce insulin resistance and type 2 diabetes by lowering belly fat. These lifestyle changes are also crucial to diabetes control in people already diagnosed.
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